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Prolactin (PRL)-PRL Receptor System Increases Cell Proliferation Involving JNK (c-Jun Amino Terminal Kinase) and AP-1 Activation: Inhibition by Glucocorticoids
Author(s) -
I. Olazabal
Publication year - 2000
Publication title -
molecular endocrinology
Language(s) - English
Resource type - Journals
eISSN - 1944-9917
pISSN - 0888-8809
DOI - 10.1210/me.14.4.564
Subject(s) - biology , prolactin receptor , c jun , signal transduction , cell growth , stat5 , endocrinology , kinase , medicine , microbiology and biotechnology , activator (genetics) , prolactin , receptor , transcription factor , hormone , biochemistry , gene

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