Impaired Glucose Metabolism in Primary Aldosteronism Is Associated With Cortisol Cosecretion
Author(s) -
Judith Gerards,
Daniel Heinrich,
Christian Adolf,
Christa Meisinger,
Wolfgang Rathmann,
Lisa Sturm,
Niirschl,
Martin Bidlingmaier,
Felix Beuschlein,
Barbara Thorand,
Annette Peters,
Martín Reincke,
Michael Roden,
Marcus Quinkler
Publication year - 2019
Publication title -
the journal of clinical endocrinology and metabolism
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 2.206
H-Index - 353
eISSN - 1945-7197
pISSN - 0021-972X
DOI - 10.1210/jc.2019-00299
Subject(s) - primary aldosteronism , medicine , endocrinology , dexamethasone suppression test , glucocorticoid , insulin resistance , fludrocortisone , impaired glucose tolerance , diabetes mellitus , pathological , body mass index , population , glucose homeostasis , homeostasis , hydrocortisone , carbohydrate metabolism , homeostatic model assessment , type 2 diabetes mellitus , dexamethasone , blood pressure , environmental health
Primary aldosteronism (PA) is associated with higher cardiovascular morbidity and metabolic risks. Recent studies report glucocorticoid cosecretion as a relevant phenotype of PA, which could contribute to associated risks, including type 2 diabetes mellitus (T2DM). The relationship between autonomous cortisol secretion (ACS) and glucose metabolism in PA has not been investigated.
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