High-Intensity Exercise Decreases IP6K1 Muscle Content and Improves Insulin Sensitivity (SI2*) in Glucose-Intolerant Individuals | Zendy

Jane Naufahu | Zendy; Bradley T. Elliott | Zendy; Anatoliy Markiv | Zendy; Petra Dunning-Foreman | Zendy; Maggie McGrady | Zendy; David Howard | Zendy; Peter Watt | Zendy; Richard Mackenzie | Zendy

Open Access

High-Intensity Exercise Decreases IP6K1 Muscle Content and Improves Insulin Sensitivity (SI2*) in Glucose-Intolerant Individuals

Author(s) -

Jane Naufahu,

Bradley T. Elliott,

Anatoliy Markiv,

Petra Dunning-Foreman,

Maggie McGrady,

David Howard,

Peter Watt,

Richard Mackenzie

Publication year - 2017

Publication title -

the journal of clinical endocrinology and metabolism

Language(s) - Uncategorized

Resource type - Journals

SCImago Journal Rank - 2.206

H-Index - 353

eISSN - 1945-7197

pISSN - 0021-972X

DOI - 10.1210/jc.2017-02019

Subject(s) - medicine , endocrinology , skeletal muscle , insulin , insulin resistance , type 2 diabetes , glucose uptake , exercise physiology , diabetes mellitus , chemistry

Insulin resistance (IR) in skeletal muscle contributes to whole body hyperglycemia and the secondary complications associated with type 2 diabetes. Inositol hexakisphosphate kinase-1 (IP6K1) may inhibit insulin-stimulated glucose transport in this tissue type.

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