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Pregnancy Augments VEGF-Stimulated In Vitro Angiogenesis and Vasodilator (NO and H2S) Production in Human Uterine Artery Endothelial Cells
Author(s) -
Hong-hai Zhang,
Jennifer C. Chen,
Lili Sheibani,
Thomas J. Lechuga,
Dong-bao Chen
Publication year - 2017
Publication title -
the journal of clinical endocrinology and metabolism
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 2.206
H-Index - 353
eISSN - 1945-7197
pISSN - 0021-972X
DOI - 10.1210/jc.2017-00437
Subject(s) - angiogenesis , enos , endocrinology , vascular endothelial growth factor , medicine , vasodilation , uterine artery , placental growth factor , endothelial stem cell , spiral artery , biology , vascular endothelial growth factor a , nitric oxide , nitric oxide synthase , placenta , in vitro , biochemistry , pregnancy , fetus , gestation , vegf receptors , genetics
Augmented uterine artery (UA) production of vasodilators, including nitric oxide (NO) and hydrogen sulfide (H2S), has been implicated in pregnancy-associated and agonist-stimulated rise in uterine blood flow that is rate-limiting to pregnancy health.

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