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Persistent Reduction of Circulating Myeloid Calcifying Cells in Acromegaly: Relevance to the Bone–Vascular Axis
Author(s) -
Gian Paolo Fadini,
Francesca Dassie,
Roberta Cappellari,
Mariasara Persano,
Saula Vigili de Kreutzenberg,
Chiara Martini,
Matteo Parolin,
Angelo Avogaro,
Roberto Vettor,
Pietro Maffei
Publication year - 2017
Publication title -
the journal of clinical endocrinology and metabolism
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 2.206
H-Index - 353
eISSN - 1945-7197
pISSN - 0021-972X
DOI - 10.1210/jc.2017-00246
Subject(s) - acromegaly , medicine , endocrinology , ectopic calcification , osteocalcin , bone remodeling , context (archaeology) , calcification , hormone , alkaline phosphatase , biology , growth hormone , paleontology , biochemistry , enzyme
Acromegaly is a systemic disease characterized by persistent bone pathology and excess cardiovascular mortality. Despite multiple concomitant risk factors, atherosclerosis does not seem to be accelerated in acromegaly.

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