Hypoxia-Dependent HIF-1 Activation Impacts on Tissue Remodeling in Graves' Ophthalmopathy—Implications for Smoking | Zendy

Gina-Eva Görtz | Zendy; Mareike Horstmann | Zendy; Barbara Aniol | Zendy; Buena Delos Reyes | Zendy; Joachim Fandrey | Zendy; Anja Eckstein | Zendy; Utta BerchnerPfannschmidt | Zendy

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Hypoxia-Dependent HIF-1 Activation Impacts on Tissue Remodeling in Graves' Ophthalmopathy—Implications for Smoking

Author(s) -

Gina-Eva Görtz,

Mareike Horstmann,

Barbara Aniol,

Buena Delos Reyes,

Joachim Fandrey,

Anja Eckstein,

Utta BerchnerPfannschmidt

Publication year - 2016

Publication title -

the journal of clinical endocrinology and metabolism

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.206

H-Index - 353

eISSN - 1945-7197

pISSN - 0021-972X

DOI - 10.1210/jc.2016-1279

Subject(s) - adipogenesis , hypoxia (environmental) , angiogenesis , medicine , endocrinology , vascular endothelial growth factor , cd31 , adiponectin , hypoxia inducible factors , inflammation , immunohistochemistry , graves' ophthalmopathy , chemistry , adipose tissue , insulin resistance , vegf receptors , obesity , biochemistry , disease , organic chemistry , oxygen , graves' disease , gene

In Graves' ophthalmopathy (GO), inflammation with tissue expansion in a closed compartment like the bony orbit and smoking may cause tissue hypoxia.

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