Resting Energy Expenditure Is Decreased in Pseudohypoparathyroidism Type 1A | Zendy

Jeffrey D. Roizen | Zendy; Jennifer A. Danzig | Zendy; Véronique Groleau | Zendy; Shana E. McCormack | Zendy; Alex Casella | Zendy; Jennifer Harrington | Zendy; Etienne Sochett | Zendy; Andrew M. Tershakovec | Zendy; Babette S. Zemel | Zendy; Virginia A. Stallings | Zendy; Michael A. Levine | Zendy

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Resting Energy Expenditure Is Decreased in Pseudohypoparathyroidism Type 1A

Author(s) -

Jeffrey D. Roizen,

Jennifer A. Danzig,

Véronique Groleau,

Shana E. McCormack,

Alex Casella,

Jennifer Harrington,

Etienne Sochett,

Andrew M. Tershakovec,

Babette S. Zemel,

Virginia A. Stallings,

Michael A. Levine

Publication year - 2015

Publication title -

the journal of clinical endocrinology and metabolism

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.206

H-Index - 353

eISSN - 1945-7197

pISSN - 0021-972X

DOI - 10.1210/jc.2015-3895

Subject(s) - resting energy expenditure , gnas complex locus , medicine , endocrine system , endocrinology , pseudohypoparathyroidism , body mass index , neurocognitive , obesity , hormone , cohort , energy expenditure , pediatrics , parathyroid hormone , biology , psychiatry , biochemistry , cognition , gene , calcium

Pseudohypoparathyroidism type 1A (PHP1A) is caused by loss-of-function mutations on the maternally inherited GNAS allele and is associated with early-onset obesity, neurocognitive defects, and resistance to multiple hormones. The role of energy intake vs central regulation of energy expenditure in the pathophysiology of obesity remains unclear.

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