Circulating Follistatin Is Liver-Derived and Regulated by the Glucagon-to-Insulin Ratio | Zendy

Jakob S. Hansen | Zendy; Sabine Rütti | Zendy; Caroline Arous | Zendy; Jens Otto Clemmesen | Zendy; Niels H. Secher | Zendy; Andrea Drescher | Zendy; Carmen GonelleGispert | Zendy; Philippe A. Halban | Zendy; Bente Klarlund Pedersen | Zendy; Cora Weigert | Zendy; Karim Bouzakri | Zendy; Peter Plomgaard | Zendy

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Circulating Follistatin Is Liver-Derived and Regulated by the Glucagon-to-Insulin Ratio

Author(s) -

Jakob S. Hansen,

Sabine Rütti,

Caroline Arous,

Jens Otto Clemmesen,

Niels H. Secher,

Andrea Drescher,

Carmen GonelleGispert,

Philippe A. Halban,

Bente Klarlund Pedersen,

Cora Weigert,

Karim Bouzakri,

Peter Plomgaard

Publication year - 2015

Publication title -

the journal of clinical endocrinology and metabolism

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.206

H-Index - 353

eISSN - 1945-7197

pISSN - 0021-972X

DOI - 10.1210/jc.2015-3668

Subject(s) - follistatin , medicine , endocrinology , glucagon , insulin , hepatocyte , biology , in vitro , biochemistry

Follistatin is a plasma protein recently reported to increase under conditions with negative energy balance, such as exercise and fasting in humans. Currently, the perception is that circulating follistatin is a result of para/autocrine actions from various tissues. The large and acute increase in circulating follistatin in response to exercise suggests that it may function as an endocrine signal.

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