Aldosterone-Signaling Defect Exacerbates Sodium Wasting in Very Preterm Neonates: The Premaldo Study | Zendy

Lætitia Martinerie | Zendy; Eric Pussard | Zendy; Nadya Yousef | Zendy; Claudine Cosson | Zendy; Ingrid Lema | Zendy; Khaled Husseini | Zendy; S. Mur | Zendy; Marc Lombès | Zendy; Pascal Boileau | Zendy

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Aldosterone-Signaling Defect Exacerbates Sodium Wasting in Very Preterm Neonates: The Premaldo Study

Author(s) -

Lætitia Martinerie,

Eric Pussard,

Nadya Yousef,

Claudine Cosson,

Ingrid Lema,

Khaled Husseini,

S. Mur,

Marc Lombès,

Pascal Boileau

Publication year - 2015

Publication title -

the journal of clinical endocrinology and metabolism

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.206

H-Index - 353

eISSN - 1945-7197

pISSN - 0021-972X

DOI - 10.1210/jc.2015-2272

Subject(s) - aldosterone , medicine , endocrinology , gestational age , renal sodium reabsorption , fractional excretion of sodium , sodium , urinary system , creatinine , plasma renin activity , kidney , renin–angiotensin system , reabsorption , pregnancy , blood pressure , chemistry , biology , genetics , organic chemistry

The neonatal period, notably in preterm infants, is characterized by high sodium wasting, implying that aldosterone, the main hormone regulating sodium reabsorption, is unable to maintain sodium homeostasis.

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