Glucagon Cell Hyperplasia and Neoplasia With and Without Glucagon Receptor Mutations
Author(s) -
Bence Sipos,
Jan Sperveslage,
Martin Anlauf,
Maike Hoffmeister,
Tobias Henopp,
Stephan Buch,
Jochen Hampe,
Achim Weber,
Pascal Hammel,
Anne Couvelard,
W Höbling,
Wolfgang Lieb,
Bernhard O. Boehm,
Günter Klöppel
Publication year - 2015
Publication title -
the journal of clinical endocrinology and metabolism
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 2.206
H-Index - 353
eISSN - 1945-7197
pISSN - 0021-972X
DOI - 10.1210/jc.2014-4405
Subject(s) - glucagon receptor , biology , missense mutation , mutation , medicine , genetics , germline mutation , endocrinology , glucagon , gene , insulin
Glucagon cell adenomatosis (GCA) was recently recognized as a multifocal hyperplastic and neoplastic disease of the glucagon cells unrelated to multiple endocrine neoplasia type 1 and von-Hippel-Lindau disease.
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