Lack of Serum Anti-Mullerian Hormone Responses After Recombinant Human Chorionic Gonadotropin Stimulation in Women With Polycystic Ovary Syndrome | Zendy

Heidi CookAndersen | Zendy; Sandy S. Chuan | Zendy; Kevin Maas | Zendy; M.A. Rosencrantz | Zendy; H. Irene Su | Zendy; Mark A. Lawson | Zendy; Helen Mason | Zendy; R. Jeffrey Chang | Zendy

Open Access

Lack of Serum Anti-Mullerian Hormone Responses After Recombinant Human Chorionic Gonadotropin Stimulation in Women With Polycystic Ovary Syndrome

Author(s) -

Heidi CookAndersen,

Sandy S. Chuan,

Kevin Maas,

M.A. Rosencrantz,

H. Irene Su,

Mark A. Lawson,

Helen Mason,

R. Jeffrey Chang

Publication year - 2014

Publication title -

the journal of clinical endocrinology and metabolism

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.206

H-Index - 353

eISSN - 1945-7197

pISSN - 0021-972X

DOI - 10.1210/jc.2014-2948

Subject(s) - polycystic ovary , endocrinology , medicine , human chorionic gonadotropin , anti müllerian hormone , androstenedione , gonadotropin , luteinizing hormone , basal (medicine) , hormone , androgen , insulin resistance , obesity , insulin

Polycystic ovary syndrome (PCOS) is an anovulatory disorder characterized by excess androgen production and increased LH secretion. Serum anti-Mullerian hormone (AMH) is also elevated in this disorder. Women with PCOS exhibit a positive correlation between AMH and LH levels and recent in vitro data demonstrate that LH can directly stimulate AMH production by granulosa cells from women with PCOS.

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