Impaired Fetoplacental Angiogenesis in Growth-Restricted Fetuses With Abnormal Umbilical Artery Doppler Velocimetry Is Mediated by Aryl Hydrocarbon Receptor Nuclear Translocator (ARNT) | Zendy

Emily Su | Zendy; Hong Xin | Zendy; Ping Yin | Zendy; Matthew T. Dyson | Zendy; John S. Coon | Zendy; Kathryn N. Farrow | Zendy; Karen K. Mestan | Zendy; Linda M. Ernst | Zendy

Open Access

Impaired Fetoplacental Angiogenesis in Growth-Restricted Fetuses With Abnormal Umbilical Artery Doppler Velocimetry Is Mediated by Aryl Hydrocarbon Receptor Nuclear Translocator (ARNT)

Author(s) -

Emily Su,

Hong Xin,

Ping Yin,

Matthew T. Dyson,

John S. Coon,

Kathryn N. Farrow,

Karen K. Mestan,

Linda M. Ernst

Publication year - 2014

Publication title -

the journal of clinical endocrinology and metabolism

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.206

H-Index - 353

eISSN - 1945-7197

pISSN - 0021-972X

DOI - 10.1210/jc.2014-2385

Subject(s) - aryl hydrocarbon receptor nuclear translocator , aryl hydrocarbon receptor , angiogenesis , fetus , chemistry , medicine , biology , gene , biochemistry , pregnancy , transcription factor , genetics

Fetal growth restriction with abnormal umbilical artery Doppler velocimetry (FGRadv), reflective of elevated fetoplacental vascular resistance, is associated with increased risks of fetal morbidity and mortality even in comparison to those of growth-restricted fetuses with normal placental blood flow. One major cause of this abnormally elevated fetoplacental vascular resistance is the aberrantly formed, thin, elongated villous vessels that are seen in FGRadv placentas.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research