Elevated Peritoneal Expression and Estrogen Regulation of Nociceptive Ion Channels in Endometriosis
Author(s) -
Erin Greaves,
Kelsey M. Grieve,
Andrew W. Horne,
Philippa T. K. Saunders
Publication year - 2014
Publication title -
the journal of clinical endocrinology and metabolism
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 2.206
H-Index - 353
eISSN - 1945-7197
pISSN - 0021-972X
DOI - 10.1210/jc.2014-2282
Subject(s) - endometriosis , trpv1 , medicine , endocrinology , estrogen , nociception , context (archaeology) , peritoneum , pelvic pain , receptor , biology , pathology , transient receptor potential channel , surgery , paleontology
Ovarian suppression is a common treatment for endometriosis-associated pelvic pain. Its exact mechanism of action is poorly understood, although it is assumed to reflect reduced production/action of estrogens.
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