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Changes in Plasma ACTH Levels and Corticotroph Tumor Size in Patients With Cushing's Disease During Long-term Treatment With the Glucocorticoid Receptor Antagonist Mifepristone
Author(s) -
Maria Fleseriu,
James W. Findling,
Christian A. Koch,
SvenMartin Schlaffer,
Michael Buchfelder,
Coleman Gross
Publication year - 2014
Publication title -
the journal of clinical endocrinology and metabolism
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 2.206
H-Index - 353
eISSN - 1945-7197
pISSN - 0021-972X
DOI - 10.1210/jc.2014-1843
Subject(s) - mifepristone , medicine , discontinuation , antiglucocorticoid , glucocorticoid , endocrinology , cushing's disease , adrenocorticotropic hormone , magnetic resonance imaging , hormone antagonist , glucocorticoid receptor , corticotropic cell , gastroenterology , anterior pituitary , antagonist , urology , receptor , hormone , disease , radiology , pregnancy , biology , genetics
Pituitary effects of long-term therapy with mifepristone, a glucocorticoid receptor antagonist, in Cushing's disease (CD) patients are not well understood.

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