Testicular Adrenal Rest Tumors Develop Independently of Long-Term Disease Control: A Longitudinal Analysis of 50 Adult Men With Congenital Adrenal Hyperplasia due to Classic 21-Hydroxylase Deficiency | Zendy

Nicole Reisch | Zendy; Marietta Rottenkolber | Zendy; Anais Greifenstein | Zendy; Nils Krone | Zendy; Heinrich Schmidt | Zendy; Martín Reincke | Zendy; HansPeter Schwarz | Zendy; Felix Beuschlein | Zendy

Open Access

Testicular Adrenal Rest Tumors Develop Independently of Long-Term Disease Control: A Longitudinal Analysis of 50 Adult Men With Congenital Adrenal Hyperplasia due to Classic 21-Hydroxylase Deficiency

Author(s) -

Nicole Reisch,

Marietta Rottenkolber,

Anais Greifenstein,

Nils Krone,

Heinrich Schmidt,

Martín Reincke,

HansPeter Schwarz,

Felix Beuschlein

Publication year - 2013

Publication title -

the journal of clinical endocrinology and metabolism

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.206

H-Index - 353

eISSN - 1945-7197

pISSN - 0021-972X

DOI - 10.1210/jc.2012-3181

Subject(s) - endocrinology , medicine , bone age , 21 hydroxylase , congenital adrenal hyperplasia , testosterone (patch) , body mass index , physiology

Testicular adrenal rest tumors (TARTs) and hypogonadotropic hypogonadism are the two most common causes for male infertility in classic 21-hydroxylase deficiency. Current hypotheses suggest the quality of disease control to be one of the main pathogenic factors for TART development.

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