Maternal Smoking and Developmental Changes in Luteinizing Hormone (LH) and the LH Receptor in the Fetal Testis | Zendy

Paul Fowler | Zendy; Siladitya Bhattacharya | Zendy; Jörg Gromoll | Zendy; Ana Monteiro | Zendy; Peter J. O’Shaughnessy | Zendy

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Maternal Smoking and Developmental Changes in Luteinizing Hormone (LH) and the LH Receptor in the Fetal Testis

Author(s) -

Paul Fowler,

Siladitya Bhattacharya,

Jörg Gromoll,

Ana Monteiro,

Peter J. O’Shaughnessy

Publication year - 2009

Publication title -

the journal of clinical endocrinology and metabolism

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.206

H-Index - 353

eISSN - 1945-7197

pISSN - 0021-972X

DOI - 10.1210/jc.2009-0994

Subject(s) - medicine , endocrinology , luteinizing hormone , testosterone (patch) , context (archaeology) , hormone , fetus , biology , pregnancy , paleontology , genetics

The LH receptor (LHCGR) drives fetal testosterone secretion, which is vital for human masculinization. Maternal smoking is associated with defective masculinization, but the relationship between smoking, tropic hormones, testosterone, and functional LHCGR expression is poorly understood.

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