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substantial evidence suggests that the expression of steroid metabolizing enzymes in endometriosis is altered, turning the ectopic endometrium into a source of 17beta-estradiol. However, whether these differences result in a net increase in local 17beta-estradiol production/activity has not been shown.

Increased Production of 17β-Estradiol in Endometriosis Lesions Is the Result of Impaired Metabolism