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Obesity is a prominent feature of Albright hereditary osteodystrophy (AHO), a disorder caused by heterozygous GNAS mutations that disrupt the stimulatory G protein alpha-subunit Galpha(s). Because Galpha(s) is paternally imprinted in certain hormone target tissues, maternal inheritance of AHO leads to multihormone resistance [pseudohypoparathyroidism type 1a (PHP1a)], whereas paternal inheritance leads to AHO alone [pseudopseudohypoparathyroidism (pseudoPHP)]. Classically, the obesity in AHO is described as occurring similarly in both conditions.

the journal of clinical endocrinology and metabolism/journal of clinical endocrinology and metabolism

Body Mass Index Differences in Pseudohypoparathyroidism Type 1a<i>Versus</i>Pseudopseudohypoparathyroidism May Implicate Paternal Imprinting of Gαs in the Development of Human Obesity

Body Mass Index Differences in Pseudohypoparathyroidism Type 1aVersusPseudopseudohypoparathyroidism May Implicate Paternal Imprinting of Gαs in the Development of Human Obesity