Effect of a p38 Mitogen-Activated Protein Kinase Inhibitor on Corneal Endothelial Cell Proliferation | Zendy

Makiko Nakahara | Zendy; Naoki Okumura | Zendy; Shinichiro Nakano | Zendy; Noriko Koizumi | Zendy

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Effect of a p38 Mitogen-Activated Protein Kinase Inhibitor on Corneal Endothelial Cell Proliferation

Author(s) -

Makiko Nakahara,

Naoki Okumura,

Shinichiro Nakano,

Noriko Koizumi

Publication year - 2018

Publication title -

investigative ophthalmology and visual science

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.935

H-Index - 218

eISSN - 1552-5783

pISSN - 0146-0404

DOI - 10.1167/iovs.18-24394

Subject(s) - mapk/erk pathway , corneal endothelium , cell growth , microbiology and biotechnology , p38 mitogen activated protein kinases , downregulation and upregulation , cell cycle , chemistry , endothelium , biology , cell , kinase , biochemistry , endocrinology , gene

We have performed clinical research on cell-based therapy for corneal endothelial decompensation since 2013. The purpose of this study was to investigate the usefulness of a p38 MAPK inhibitor for promoting proliferation of human corneal endothelial cells (HCECs).

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