Ecel1 Knockdown With an AAV2-Mediated CRISPR/Cas9 System Promotes Optic Nerve Damage-Induced RGC Death in the Mouse Retina | Zendy

Kota Sato | Zendy; Yukihiro Shiga | Zendy; Yurika Nakagawa-Saito | Zendy; Kosuke Fujita | Zendy; Koji M. Nishiguchi | Zendy; Hiroshi Tawarayama | Zendy; Namie Murayama | Zendy; Shigeto Maekawa | Zendy; Takeshi Yabana | Zendy; Kazuko Omodaka | Zendy; Shota Katayama | Zendy; Qiwei Feng | Zendy; Satoru Tsuda | Zendy; Toru Nakazawa | Zendy

Open Access

Ecel1 Knockdown With an AAV2-Mediated CRISPR/Cas9 System Promotes Optic Nerve Damage-Induced RGC Death in the Mouse Retina

Author(s) -

Kota Sato,

Yukihiro Shiga,

Yurika Nakagawa-Saito,

Kosuke Fujita,

Koji M. Nishiguchi,

Hiroshi Tawarayama,

Namie Murayama,

Shigeto Maekawa,

Takeshi Yabana,

Kazuko Omodaka,

Shota Katayama,

Qiwei Feng,

Satoru Tsuda,

Toru Nakazawa

Publication year - 2018

Publication title -

investigative ophthalmology and visual science

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.935

H-Index - 218

eISSN - 1552-5783

pISSN - 0146-0404

DOI - 10.1167/iovs.18-23784

Subject(s) - optic nerve , retinal ganglion cell , retina , gene knockdown , biology , excitotoxicity , neuroprotection , microbiology and biotechnology , medicine , neuroscience , anatomy , glutamate receptor , genetics , apoptosis , receptor

To assess the therapeutic potential of endothelin-converting enzyme-like 1 (Ecel1) in a mouse model of optic nerve crush.

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