z-logo
open-access-imgOpen Access
Netrin-4 Mediates Corneal Hemangiogenesis but Not Lymphangiogenesis in the Mouse-Model of Suture-Induced Neovascularization
Author(s) -
Anna-Karina B. Maier,
S. Klein,
Norbert Kociok,
Aline I. Riechardt,
Enken Gundlach,
Nadine Reichhart,
Olaf Strauß,
Antonia M. Joussen
Publication year - 2017
Publication title -
investigative ophthalmology and visual science
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.935
H-Index - 218
eISSN - 1552-5783
pISSN - 0146-0404
DOI - 10.1167/iovs.16-19249
Subject(s) - lymphangiogenesis , corneal neovascularization , netrin , fibrous joint , neovascularization , medicine , ophthalmology , microbiology and biotechnology , cancer research , angiogenesis , anatomy , biology , cancer , metastasis , axon , axon guidance
Netrin-4, a secreted protein, is found in the basement membrane of blood vessels and acts as a key regulator of angiogenesis. Here we investigated the role of Netrin-4 in the mouse-model of suture-induced corneal hem- and lymphangiogenesis.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.
Having issues? You can contact us here
Accelerating Research

Address

John Eccles House
Robert Robinson Avenue,
Oxford Science Park, Oxford
OX4 4GP, United Kingdom