Deletion of Aryl Hydrocarbon Receptor AHR in Mice Leads to Subretinal Accumulation of Microglia and RPE Atrophy | Zendy

SooYoung Kim | Zendy; Hyun-Jin Yang | Zendy; Yi-Sheng Chang | Zendy; Jung-Woong Kim | Zendy; Matthew Brooks | Zendy; Emily Y. Chew | Zendy; Wai T. Wong | Zendy; Robert N. Fariss | Zendy; Rivka A. Rachel | Zendy; Tiziana Cogliati | Zendy; Haohua Qian | Zendy; Anand Swaroop | Zendy

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Deletion of Aryl Hydrocarbon Receptor AHR in Mice Leads to Subretinal Accumulation of Microglia and RPE Atrophy

Author(s) -

SooYoung Kim,

Hyun-Jin Yang,

Yi-Sheng Chang,

Jung-Woong Kim,

Matthew Brooks,

Emily Y. Chew,

Wai T. Wong,

Robert N. Fariss,

Rivka A. Rachel,

Tiziana Cogliati,

Haohua Qian,

Anand Swaroop

Publication year - 2014

Publication title -

investigative ophthalmology and visual science

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.935

H-Index - 218

eISSN - 1552-5783

pISSN - 0146-0404

DOI - 10.1167/iovs.14-15091

Subject(s) - aryl hydrocarbon receptor , microglia , retina , glial fibrillary acidic protein , retinal , electroretinography , biology , microbiology and biotechnology , pathology , endocrinology , immunohistochemistry , inflammation , neuroscience , medicine , immunology , transcription factor , biochemistry , gene

The aryl hydrocarbon receptor (AHR) is a ligand-activated nuclear receptor that regulates cellular response to environmental signals, including UV and blue wavelength light. This study was undertaken to elucidate AHR function in retinal homeostasis.

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