Effect of Hypoxic Stress–Activated Polo-like Kinase 3 on Corneal Epithelial Wound Healing | Zendy

Jiawei Lu | Zendy; Ling Wang | Zendy; Wei Dai | Zendy; Luo Lu | Zendy

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Effect of Hypoxic Stress–Activated Polo-like Kinase 3 on Corneal Epithelial Wound Healing

Author(s) -

Jiawei Lu,

Ling Wang,

Wei Dai,

Luo Lu

Publication year - 2010

Publication title -

investigative ophthalmology and visual science

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.935

H-Index - 218

eISSN - 1552-5783

pISSN - 0146-0404

DOI - 10.1167/iovs.10-5501

Subject(s) - wound healing , corneal epithelium , oxidative stress , cornea , kinase , hypoxia (environmental) , chemistry , andrology , medicine , microbiology and biotechnology , biology , immunology , ophthalmology , biochemistry , organic chemistry , oxygen

Hypoxia/reoxygenation conditions can generate oxidative stresses resulting in the suppression of cell proliferation and the delay of corneal epithelial wound healing. The purpose of this study was to investigate the cellular mechanism involving the role of the stress-responsive Polo-like kinase 3 (Plk3) in hypoxic stress-induced delay of corneal epithelial wound healing.

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