Triamcinolone Reduces Neovascularization, Capillary Density and IGF-1 Receptor Phosphorylation in a Model of Oxygen-Induced Retinopathy | Zendy

M. Elizabeth Hartnett | Zendy; David Martiniuk | Zendy; Yuta Saito | Zendy; Pete Geisen | Zendy; Lynda J. Peterson | Zendy; Janet R. McColm | Zendy

Open Access

Triamcinolone Reduces Neovascularization, Capillary Density and IGF-1 Receptor Phosphorylation in a Model of Oxygen-Induced Retinopathy

Author(s) -

M. Elizabeth Hartnett,

David Martiniuk,

Yuta Saito,

Pete Geisen,

Lynda J. Peterson,

Janet R. McColm

Publication year - 2006

Publication title -

investigative ophthalmology and visual science

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.935

H-Index - 218

eISSN - 1552-5783

pISSN - 0146-0404

DOI - 10.1167/iovs.06-0450

Subject(s) - triamcinolone acetonide , retinal , trypan blue , neovascularization , retina , vascular endothelial growth factor , endocrinology , medicine , in vivo , retinopathy , ophthalmology , apoptosis , chemistry , angiogenesis , biology , biochemistry , diabetes mellitus , vegf receptors , microbiology and biotechnology , neuroscience

To study the effects of intravitreous triamcinolone acetonide (TA) on neovascularization (NV), capillary density, and retinal endothelial cell (REC) viability in a model of oxygen-induced retinopathy (OIR).

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