Open AccessMolecular Targeting of Antiangiogenic Factor 16K hPRL Inhibits Oxygen-Induced Retinopathy in Mice
Author(s) -
Hao Pan,
Ngoc-Quynh-Nhu Nguyen,
Hiroshi Yoshida,
Frauke Bentzien,
Lynn C. Shaw,
Françoise RentierDelrue,
Joseph Martial,
Richard I. Weiner,
Ingrid Struman,
Maria B. Grant
Publication year - 2004
Publication title -
investigative ophthalmology and visual science
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.935
H-Index - 218
eISSN - 1552-5783
pISSN - 0146-0404
DOI - 10.1167/iovs.03-1001
Subject(s) - retinal , apoptosis , neovascularization , endocrinology , diabetic retinopathy , medicine , retina , biology , angiogenesis , ophthalmology , biochemistry , diabetes mellitus , neuroscience
To examine the ability and mechanism of the 16 kDa N-terminal fragment of human prolactin (16K hPRL) in the inhibition of abnormal retinal neovascularization.
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