Limited Ability to Activate Protein C Confers Left Atrial Endocardium A Thrombogenic Phenotype | Zendy

Jorge Cerveró | Zendy; Ramón Montes | Zendy; Francisco España | Zendy; Charles T. Esmon | Zendy; José Hermida | Zendy

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Limited Ability to Activate Protein C Confers Left Atrial Endocardium A Thrombogenic Phenotype

Author(s) -

Jorge Cerveró,

Ramón Montes,

Francisco España,

Charles T. Esmon,

José Hermida

Publication year - 2011

Publication title -

stroke

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.397

H-Index - 319

eISSN - 1524-4628

pISSN - 0039-2499

DOI - 10.1161/strokeaha.111.614420

Subject(s) - thrombomodulin , endocardium , thrombogenicity , medicine , protein c , atrial fibrillation , cardiology , receptor , stroke (engine) , endocrinology , thrombin , thrombosis , platelet , mechanical engineering , engineering

Atrial fibrillation is the most important risk factor for cardioembolic stroke. Thrombi form in the left atrial appendage rather than in the right. The causes of this different thrombogenicity are not well-understood. The goal herein was to compare the activation of the anticoagulant protein C and the thrombomodulin and endothelial protein C receptor/activated protein C receptor expression on the endocardium between right and left atria.

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