Dalfampridine Improves Sensorimotor Function in Rats With Chronic Deficits After Middle Cerebral Artery Occlusion | Zendy

Jennifer F. Iaci | Zendy; Tom J. Parry | Zendy; Zhihong Huang | Zendy; Seth P. Finklestein | Zendy; JingMei Ren | Zendy; Dana K. Barrile | Zendy; Matthew D. Davenport | Zendy; Rui Wu | Zendy; Andrew R. Blight | Zendy; Anthony O. Caggiano | Zendy

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Dalfampridine Improves Sensorimotor Function in Rats With Chronic Deficits After Middle Cerebral Artery Occlusion

Author(s) -

Jennifer F. Iaci,

Tom J. Parry,

Zhihong Huang,

Seth P. Finklestein,

JingMei Ren,

Dana K. Barrile,

Matthew D. Davenport,

Rui Wu,

Andrew R. Blight,

Anthony O. Caggiano

Publication year - 2013

Publication title -

stroke

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.397

H-Index - 319

eISSN - 1524-4628

pISSN - 0039-2499

DOI - 10.1161/strokeaha.111.000147

Subject(s) - medicine , middle cerebral artery , stroke (engine) , occlusion , anesthesia , physical medicine and rehabilitation , cardiology , ischemia , mechanical engineering , engineering

Stroke survivors often have permanent deficits that are only partially addressed by physical therapy. This study evaluated the effects of dalfampridine, a potassium channel blocker, on persistent sensorimotor deficits in rats with treatment initiated 4 or 8 weeks after stroke.

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