Osteopontin Reduced Hypoxia–Ischemia Neonatal Brain Injury by Suppression of Apoptosis in a Rat Pup Model | Zendy

Wanqiu Chen | Zendy; Qingyi Ma | Zendy; Hidenori Suzuki | Zendy; Richard E. Hartman | Zendy; Jiping Tang | Zendy; John H. Zhang | Zendy

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Osteopontin Reduced Hypoxia–Ischemia Neonatal Brain Injury by Suppression of Apoptosis in a Rat Pup Model

Author(s) -

Wanqiu Chen,

Qingyi Ma,

Hidenori Suzuki,

Richard E. Hartman,

Jiping Tang,

John H. Zhang

Publication year - 2011

Publication title -

stroke

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.397

H-Index - 319

eISSN - 1524-4628

pISSN - 0039-2499

DOI - 10.1161/strokeaha.110.599118

Subject(s) - neuroprotection , osteopontin , medicine , hypoxia (environmental) , ischemia , morris water navigation task , endogeny , apoptosis , brain damage , anesthesia , pharmacology , endocrinology , pathology , hippocampus , biology , biochemistry , chemistry , organic chemistry , oxygen

Osteopontin (OPN) is neuroprotective in ischemic brain injuries in adult experimental models; therefore, we hypothesized that OPN would provide neuroprotection and improve long-term neurological function in the immature brain after hypoxic-ischemic (HI) injury.

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