Activation of Epsilon Protein Kinase C-Mediated Anti-Apoptosis Is Involved in Rapid Tolerance Induced by Electroacupuncture Pretreatment Through Cannabinoid Receptor Type 1 | Zendy

Qiang Wang | Zendy; Xuying Li | Zendy; Yanke Chen | Zendy; Feng Wang | Zendy; Qianzi Yang | Zendy; Shaoyang Chen | Zendy; Yuyuan Min | Zendy; Xin Li | Zendy; Lize Xiong | Zendy

Open Access

Activation of Epsilon Protein Kinase C-Mediated Anti-Apoptosis Is Involved in Rapid Tolerance Induced by Electroacupuncture Pretreatment Through Cannabinoid Receptor Type 1

Author(s) -

Qiang Wang,

Xuying Li,

Yanke Chen,

Feng Wang,

Qianzi Yang,

Shaoyang Chen,

Yuyuan Min,

Xin Li,

Lize Xiong

Publication year - 2010

Publication title -

stroke

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.397

H-Index - 319

eISSN - 1524-4628

pISSN - 0039-2499

DOI - 10.1161/strokeaha.110.597336

Subject(s) - neuroprotection , medicine , cannabinoid , pharmacology , ischemia , protein kinase c , cannabinoid receptor , brain ischemia , receptor , endocrinology , anesthesia , kinase , biology , antagonist , microbiology and biotechnology

Our previous study has demonstrated that the rapid tolerance to cerebral ischemia by electroacupuncture (EA) pretreatment was possibly mediated through an endocannabinoid system-related mechanism. The purpose of this study was to investigate whether activation of epsilon protein kinase C (εPKC) was involved in EA pretreatment-induced neuroprotection via cannabinoid receptor type 1 in a rat model of transient focal cerebral ischemia.

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