Enhanced Capillary Amyloid Angiopathy-Associated Pathology in Tg-SwDI Mice With Deleted Nitric Oxide Synthase 2 | Zendy

William E. Van Nostrand | Zendy; Feng Xu | Zendy; Annemieke J.M. Rozemüller | Zendy; Carol A. Colton | Zendy

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Enhanced Capillary Amyloid Angiopathy-Associated Pathology in Tg-SwDI Mice With Deleted Nitric Oxide Synthase 2

Author(s) -

William E. Van Nostrand,

Feng Xu,

Annemieke J.M. Rozemüller,

Carol A. Colton

Publication year - 2010

Publication title -

stroke

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.397

H-Index - 319

eISSN - 1524-4628

pISSN - 0039-2499

DOI - 10.1161/strokeaha.110.595272

Subject(s) - cerebral amyloid angiopathy , neuroinflammation , microglia , amyloid (mycology) , pathology , medicine , nitric oxide synthase , amyloid precursor protein , senile plaques , amyloidosis , angiopathy , immunohistochemistry , nitric oxide , endocrinology , alzheimer's disease , inflammation , dementia , diabetes mellitus , disease

Cerebral amyloid angiopathy Type 1 is characterized by amyloid β protein deposition along cerebral capillaries and is accompanied by perivascular neuroinflammation and accumulation of phospho-tau protein. Tg-SwDI mice recapitulate capillary amyloid deposition and associated neuroinflammation but lack accumulation of perivascular phospho-tau protein.

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