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Oxidative Stress After Thrombolysis-Induced Reperfusion in Human Stroke
Author(s) -
Carmen Carrillo Domínguez,
Pilar Delgado,
Ángel VilchesArenas,
Pilar Martı́n-Gallán,
Marc Ribó,
Estevo Santamarina,
Carlos A. Molina,
Natália Corbeto,
Victor RodríguezSureda,
Anna Rosell,
José ÁlvarezSabín,
Joan Montaner
Publication year - 2010
Publication title -
stroke
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 3.397
H-Index - 319
eISSN - 1524-4628
pISSN - 0039-2499
DOI - 10.1161/strokeaha.109.571935
Subject(s) - medicine , thrombolysis , oxidative stress , stroke (engine) , ischemic stroke , cardiology , ischemia , myocardial infarction , mechanical engineering , engineering
Animal models of transient ischemia suggest that oxygen-derived free radicals produced on reperfusion of ischemic brain could constitute the main cause of reperfusion injury. We aimed to determine the presence and role of lipid peroxidation and protein oxidation-related molecules after tissue plasminogen activator-induced recanalization in human stroke.

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