Plasma β-Amyloid 1-40 Is Associated With the Diffuse Small Vessel Disease Subtype | Zendy

Meritxell Gomis | Zendy; Tomás Sobrino | Zendy; Ángel Ois | Zendy; Mónica Millán | Zendy; Ana Rodríguez-Campello | Zendy; Natàlia Pérez de la Ossa | Zendy; Raquel RodríguezGonzález | Zendy; Jordi Jiménez-Conde | Zendy; Elisa CuadradoGodia | Zendy; Jaume Roquer | Zendy; Antoni Dávalos | Zendy

AI Assistant Blog Pricing

Home ZAIA Blog

Open Access

Plasma β-Amyloid 1-40 Is Associated With the Diffuse Small Vessel Disease Subtype

Author(s) -

Meritxell Gomis,

Tomás Sobrino,

Ángel Ois,

Mónica Millán,

Ana Rodríguez-Campello,

Natàlia Pérez de la Ossa,

Raquel RodríguezGonzález,

Jordi Jiménez-Conde,

Elisa CuadradoGodia,

Jaume Roquer,

Antoni Dávalos

Publication year - 2009

Publication title -

stroke

Language(s) - English

Resource type - Journals

eISSN - 1524-4628

pISSN - 0039-2499

DOI - 10.1161/strokeaha.109.559641

Subject(s) - medicine , amyloid (mycology) , disease , pathology , stroke (engine) , engineering , mechanical engineering

The underlying mechanisms of small vessel disease (SVD) subtypes are diffuse arteriopathy (diffuse-SVD) or microatheroma (focal-SVD). Endothelial dysfunction by beta-amyloid peptide (Abeta) deposition has been associated with lacunar infarcts and leukoaraiosis, but its specific relationship with SVD subtypes is unknown. We hypothesized that plasma Abeta levels can play a different role in SVD subtypes in patients with acute lacunar stroke.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research