BNIP3 Upregulation and EndoG Translocation in Delayed Neuronal Death in Stroke and in Hypoxia | Zendy

Zhengfeng Zhang | Zendy; Xuefen Yang | Zendy; Surong Zhang | Zendy; Xiuli Ma | Zendy; Jiming Kong | Zendy

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BNIP3 Upregulation and EndoG Translocation in Delayed Neuronal Death in Stroke and in Hypoxia

Author(s) -

Zhengfeng Zhang,

Xuefen Yang,

Surong Zhang,

Xiuli Ma,

Jiming Kong

Publication year - 2007

Publication title -

stroke

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.397

H-Index - 319

eISSN - 1524-4628

pISSN - 0039-2499

DOI - 10.1161/strokeaha.106.475129

Subject(s) - medicine , hypoxia (environmental) , downregulation and upregulation , stroke (engine) , neuroscience , cardiology , oxygen , gene , biochemistry , chemistry , organic chemistry , biology , mechanical engineering , engineering

Delayed neuronal death is a hallmark feature of stroke and the primary target of neuroprotective strategies. Caspase-independent apoptosis pathways are suggested as a mechanism for the delayed neuronal injury. Here we test the hypothesis that one of the caspase-independent apoptosis pathways is activated by BNIP3 and mediated by EndoG.

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