Targeted hsp70.1 Disruption Increases Infarction Volume After Focal Cerebral Ischemia in Mice | Zendy

SeungHoon Lee | Zendy; Manho Kim | Zendy; ByungWoo Yoon | Zendy; YoungJu Kim | Zendy; Sung-Jin Ma | Zendy; JaeKyu Roh | Zendy; JaeSeon Lee | Zendy; Jeong-Sun Seo | Zendy

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Targeted hsp70.1 Disruption Increases Infarction Volume After Focal Cerebral Ischemia in Mice

Author(s) -

SeungHoon Lee,

Manho Kim,

ByungWoo Yoon,

YoungJu Kim,

Sung-Jin Ma,

JaeKyu Roh,

JaeSeon Lee,

Jeong-Sun Seo

Publication year - 2001

Publication title -

stroke

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.397

H-Index - 319

eISSN - 1524-4628

pISSN - 0039-2499

DOI - 10.1161/hs1201.099604

Subject(s) - hsp70 , knockout mouse , ischemia , medicine , penumbra , heat shock protein , blot , infarction , immunohistochemistry , cerebral infarction , middle cerebral artery , brain ischemia , pathology , endocrinology , biology , receptor , biochemistry , myocardial infarction , gene

Heat-shock proteins (HSPs) are highly conserved proteins that are induced by a variety of stresses. HSP70 is a 70-kDa HSP family known to have cytoprotective effects against various insults. The role of HSP70 in cerebral ischemia remains to be elucidated in vivo.

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