Prolonged Cerebral Transit Time in CADASIL | Zendy

Martin Liebetrau | Zendy; Jürgen Herzog | Zendy; Christian U.A. Kloss | Zendy; Gerhard F. Hamann | Zendy; Martin Dichgans | Zendy

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Prolonged Cerebral Transit Time in CADASIL

Author(s) -

Martin Liebetrau,

Jürgen Herzog,

Christian U.A. Kloss,

Gerhard F. Hamann,

Martin Dichgans

Publication year - 2002

Publication title -

stroke

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.397

H-Index - 319

eISSN - 1524-4628

pISSN - 0039-2499

DOI - 10.1161/hs0202.102949

Subject(s) - cadasil , medicine , leukoencephalopathy , angiopathy , cardiology , microangiopathy , stroke (engine) , cerebral arteries , vascular disease , posterior cerebral artery , middle cerebral artery , disease , ischemia , endocrinology , diabetes mellitus , mechanical engineering , engineering

Cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) is a hereditary angiopathy caused by mutations in Notch3. Cerebral microvessels show an accumulation of granular osmophilic material in the vicinity of degenerating vascular smooth muscle cells. In this study, we measured the arteriovenous cerebral transit time (CTT) to identify changes related to the microangiopathy in CADASIL.

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