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The number of CD4(+)CD28(null) (CD28(null)) T cells, a unique subset of T lymphocytes with proinflammatory and cell-lytic phenotype, increases markedly in patients with acute coronary syndrome (ACS). ACS patients harboring high numbers of CD28(null) T cells have increased risk of recurrent severe acute coronary events and unfavorable prognosis. The mechanisms that govern the increase in CD28(null) T cells in ACS remain elusive. We investigated whether apoptosis pathways regulating T-cell homeostasis are perturbed in CD28(null) T cells in ACS.

circulation

Proteasome-Mediated Reduction in Proapoptotic Molecule Bim Renders CD4<sup>+</sup>CD28<sup>null</sup>T Cells Resistant to Apoptosis in Acute Coronary Syndrome

Proteasome-Mediated Reduction in Proapoptotic Molecule Bim Renders CD4+CD28nullT Cells Resistant to Apoptosis in Acute Coronary Syndrome

Proteasome-Mediated Reduction in Proapoptotic Molecule Bim Renders CD4⁺CD28^nullT Cells Resistant to Apoptosis in Acute Coronary Syndrome