Dual-Specificity Phosphatase 3 Deficiency or Inhibition Limits Platelet Activation and Arterial Thrombosis | Zendy

Lucia Musumeci | Zendy; Marijke J. E. Kuijpers | Zendy; Karen Gilio | Zendy; Alexandre Hego | Zendy; Emilie Théâtre | Zendy; L.F.A. Maurissen | Zendy; Maud Vandereyken | Zendy; Cátia V. Diogo | Zendy; Christelle Lecut | Zendy; William Guilmain | Zendy; Ekaterina V. Bobkova | Zendy; Johannes A. Eble | Zendy; Russell Dahl | Zendy; Pierre Drion | Zendy; Justin Rascon | Zendy; Yalda Mostofi | Zendy; Qiang Yu | Zendy; Eduard Sergienko | Zendy; Thomas D.Y. Chung | Zendy; Marc Thiry | Zendy; Yotis A. Senis | Zendy; Michel Moutschen | Zendy; Tomas Mustelin | Zendy; Patrizio Lancellotti | Zendy; Johan W. M. Heemskerk | Zendy; Lutz Tautz | Zendy; Cécile Oury | Zendy; Souad Rahmouni | Zendy

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Dual-Specificity Phosphatase 3 Deficiency or Inhibition Limits Platelet Activation and Arterial Thrombosis

Author(s) -

Lucia Musumeci,

Marijke J. E. Kuijpers,

Karen Gilio,

Alexandre Hego,

Emilie Théâtre,

L.F.A. Maurissen,

Maud Vandereyken,

Cátia V. Diogo,

Christelle Lecut,

William Guilmain,

Ekaterina V. Bobkova,

Johannes A. Eble,

Russell Dahl,

Pierre Drion,

Justin Rascon,

Yalda Mostofi,

Qiang Yu,

Eduard Sergienko,

Thomas D.Y. Chung,

Marc Thiry,

Yotis A. Senis,

Michel Moutschen,

Tomas Mustelin,

Patrizio Lancellotti,

Johan W. M. Heemskerk,

Lutz Tautz,

Cécile Oury,

Souad Rahmouni

Publication year - 2014

Publication title -

circulation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 7.795

H-Index - 607

eISSN - 1524-4539

pISSN - 0009-7322

DOI - 10.1161/circulationaha.114.010186

Subject(s) - medicine , thrombosis , platelet activation , platelet , cardiology

A limitation of current antiplatelet therapies is their inability to separate thrombotic events from bleeding occurrences. A better understanding of the molecular mechanisms leading to platelet activation is important for the development of improved therapies. Recently, protein tyrosine phosphatases have emerged as critical regulators of platelet function.

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