Cellular and Molecular Mechanisms of Atrial Arrhythmogenesis in Patients With Paroxysmal Atrial Fibrillation | Zendy

Niels Voigt | Zendy; Jordi Heijman | Zendy; Qiongling Wang | Zendy; David Y. Chiang | Zendy; Na Li | Zendy; Matthias Karck | Zendy; Xander H.T. Wehrens | Zendy; Stanley Nattel | Zendy; Dobromir Dobrev | Zendy

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Cellular and Molecular Mechanisms of Atrial Arrhythmogenesis in Patients With Paroxysmal Atrial Fibrillation

Author(s) -

Niels Voigt,

Jordi Heijman,

Qiongling Wang,

David Y. Chiang,

Na Li,

Matthias Karck,

Xander H.T. Wehrens,

Stanley Nattel,

Dobromir Dobrev

Publication year - 2013

Publication title -

circulation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 7.795

H-Index - 607

eISSN - 1524-4539

pISSN - 0009-7322

DOI - 10.1161/circulationaha.113.006641

Subject(s) - medicine , paroxysmal atrial fibrillation , atrial fibrillation , cardiology , p wave

Electrical, structural, and Ca2+ -handling remodeling contribute to the perpetuation/progression of atrial fibrillation (AF). Recent evidence has suggested a role for spontaneous sarcoplasmic reticulum Ca2+ -release events in long-standing persistent AF, but the occurrence and mechanisms of sarcoplasmic reticulum Ca2+ -release events in paroxysmal AF (pAF) are unknown.

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