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Mitral Valve Injury After Radiofrequency Ablation for Wolff-Parkinson-White Syndrome
Author(s) -
Juan G. Penaranda Canal,
Maurice EnriquezSarano,
Samuel J. Asirvatham,
Thomas M. Munger,
Paul A. Friedman,
Rakesh M. Suri
Publication year - 2013
Publication title -
circulation
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 7.795
H-Index - 607
eISSN - 1524-4539
pISSN - 0009-7322
DOI - 10.1161/circulationaha.113.002711
Subject(s) - medicine , white (mutation) , surgery , biochemistry , gene , chemistry
A 39-year-old athletic female with symptomatic Wolff-Parkinson-White syndrome underwent an electrophysiological study and subsequent ablation of orthodromic reentrant tachycardias using left lateral, posterolateral, and posteroseptal accessory pathways. Ablation was performed with 7F medium- and large-curl Safire catheters using the retrograde aortic approach. Eighteen radiofrequency lesions were delivered at the mitral valve annulus (50 W, 60°, 10–60 seconds), and 8 were delivered in the coronary sinus, with weak persistence of antegrade conduction through the posteroseptal pathway. Two weeks later, she presented to the emergency room with a new episode of supraventricular tachycardia, She had been having dyspnea since the ablation procedure. A transthoracic echocardiogram revealed severe mitral regurgitation with a regurgitant volume of 66 ml and estimated regurgitant orifice of 39 mm2 (Figure [C, F, and G]); the left atrium was enlarged with a volume index of 36 ml/m2. Transesophageal echocardiogram revealed a ruptured chord to the anterior leaflet adjacent to the posteromedial commissure (Figure [E]). Given the symptoms and severity of the mitral valve regurgitation, the decision …

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