Lysozyme M–Positive Monocytes Mediate Angiotensin II–Induced Arterial Hypertension and Vascular Dysfunction | Zendy

Philip Wenzel | Zendy; Maike Knorr | Zendy; Sabine Kossmann | Zendy; Jan A. Stratmann | Zendy; Michael Hausding | Zendy; Swenja Schuhmacher | Zendy; Susanne Karbach | Zendy; Melanie Schwenk | Zendy; Nir Yogev | Zendy; Eberhard Schulz | Zendy; Matthias Oelze | Zendy; Stephan Grabbe | Zendy; Helmut Jonuleit | Zendy; Christian Becker | Zendy; Andreas Daiber | Zendy; Ari Waisman | Zendy; Thomas Münzel | Zendy

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Lysozyme M–Positive Monocytes Mediate Angiotensin II–Induced Arterial Hypertension and Vascular Dysfunction

Author(s) -

Philip Wenzel,

Maike Knorr,

Sabine Kossmann,

Jan A. Stratmann,

Michael Hausding,

Swenja Schuhmacher,

Susanne Karbach,

Melanie Schwenk,

Nir Yogev,

Eberhard Schulz,

Matthias Oelze,

Stephan Grabbe,

Helmut Jonuleit,

Christian Becker,

Andreas Daiber,

Ari Waisman,

Thomas Münzel

Publication year - 2011

Publication title -

circulation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 7.795

H-Index - 607

eISSN - 1524-4539

pISSN - 0009-7322

DOI - 10.1161/circulationaha.111.034470

Subject(s) - medicine , angiotensin ii , pathophysiology of hypertension , lysozyme , endothelial dysfunction , cardiology , vascular smooth muscle , vascular disease , endocrinology , blood pressure , smooth muscle , biochemistry , chemistry

Angiotensin II (ATII), a potent vasoconstrictor, causes hypertension, promotes infiltration of myelomonocytic cells into the vessel wall, and stimulates both vascular and inflammatory cell NADPH oxidases. The predominant source of reactive oxygen species, eg, vascular (endothelial, smooth muscle, adventitial) versus phagocytic NADPH oxidase, and the role of myelomonocytic cells in mediating arterial hypertension have not been defined yet.

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