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Catheter Ablation of Ventricular Tachycardia
Author(s) -
Roderick Tung,
Noel G. Boyle,
Kalyanam Shivkumar
Publication year - 2010
Publication title -
circulation
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 7.795
H-Index - 607
eISSN - 1524-4539
pISSN - 0009-7322
DOI - 10.1161/circulationaha.110.963371
Subject(s) - medicine , cardiology , tachycardia , catheter ablation , ventricular tachycardia , ablation
Ventricular tachycardia (VT) is an abnormal rapid heart rhythm originating from the lower pumping chambers of the heart (ventricles). The normal heart usually beats between 60 and 100 times per minute, with the atria contracting first, followed by the ventricles in a synchronized fashion. In VT, the ventricles beat at a rapid rate, typically from 120 to 300 beats per minute, and are no longer coordinated with the atria. The controlled contraction of the ventricles is important for the heart to pump blood to the brain and the rest of the body and to maintain a normal blood pressure. Abnormal and fast rhythms from the ventricle may impair the ability of the pump to supply blood to the brain and the rest of the body as a result of the rapid rate and weak contractions. This may result in palpitations (a feeling of rapid or abnormal heart beat), dizziness, lightheadedness, or syncope (loss of consciousness). If the heart rate increases to more than 300 beats per minute and becomes totally uncoordinated, this is usually called ventricular fibrillation (VF), which will cause sudden cardiac death. VT occurs most commonly in patients with weakened heart muscle (cardiomyopathy) or when scar tissue develops in the heart. In patients with coronary artery disease (blockage of blood vessels on the surface of the heart), this scar is the result of a prior heart attack (myocardial infarction) when the muscle dies as a result of a blockage in blood flow. Scar, or fibrosis, can interfere with the normal electrical impulse in the heart, leading to a short-circuiting of the rhythm, called reentry. VT can also occur in patients with normal hearts by a different mechanism whereby the electric conduction is overly excitable, like a muscle twitch. Sudden cardiac death causes about 450 000 fatalities each year in the United States alone. It is most commonly caused by VT deteriorating into VF, which is fatal within a few minutes if not defibrillated (shocked) back to a normal rhythm. (Figure 1A) Defibrillation may be accomplished by an automated external defibrillator or an implantable cardioverter-defibrillator (ICD). It is important to distinguish VT and VF, which are electric problems of the heart, from a heart attack, which is due to the sudden blockage of an artery. Heart attacks are treated with clot-busting drugs, balloon angioplasty, or stents. Sometimes, VT and VF are seen in that setting and are treated with electric shocks and drugs. The treatment of abnormal rhythms is discussed below.

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