Transmural Differences in Myocardial Contraction in Long-QT Syndrome | Zendy

Kristina H. Haugaa | Zendy; Jan Amlie | Zendy; Knut Erik Berge | Zendy; Trond P. Leren | Zendy; Otto A. Smiseth | Zendy; Thor Edvardsen | Zendy

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Transmural Differences in Myocardial Contraction in Long-QT Syndrome

Author(s) -

Kristina H. Haugaa,

Jan Amlie,

Knut Erik Berge,

Trond P. Leren,

Otto A. Smiseth,

Thor Edvardsen

Publication year - 2010

Publication title -

circulation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 7.795

H-Index - 607

eISSN - 1524-4539

pISSN - 0009-7322

DOI - 10.1161/circulationaha.110.960377

Subject(s) - medicine , asymptomatic , cardiology , contraction (grammar) , long qt syndrome , electrocardiography , heart failure , qt interval

Long-QT syndrome (LQTS) is characterized by prolonged myocardial action potential duration. The longest action potential duration is reported in the endomyocardium and midmyocardium. Prolonged action potential duration in LQTS may cause prolonged cardiac contraction, which can be assessed by strain echocardiography. We hypothesized that myocardial contraction is most prolonged in subendocardial myofibers in LQTS patients and that inhomogeneous transmural contraction is related to the risk of spontaneous arrhythmia.

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