Mechanisms of Enhanced β-Adrenergic Reserve From Cardiac Resynchronization Therapy | Zendy

Khalid Chakir | Zendy; Samantapudi K. Daya | Zendy; Takeshi Aiba | Zendy; Richard S. Tunin | Zendy; Veronica L. Dimaano | Zendy; Theodore P. Abraham | Zendy; Kathryn M. Jaques | Zendy; Edwin W. Lai | Zendy; Karel Pacák | Zendy; Wei-Zhong Zhu | Zendy; Rui-Ping Xiao | Zendy; Gordon F. Tomaselli | Zendy; David A. Kass | Zendy

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Mechanisms of Enhanced β-Adrenergic Reserve From Cardiac Resynchronization Therapy

Author(s) -

Khalid Chakir,

Samantapudi K. Daya,

Takeshi Aiba,

Richard S. Tunin,

Veronica L. Dimaano,

Theodore P. Abraham,

Kathryn M. Jaques,

Edwin W. Lai,

Karel Pacák,

Wei-Zhong Zhu,

Rui-Ping Xiao,

Gordon F. Tomaselli,

David A. Kass

Publication year - 2009

Publication title -

circulation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 7.795

H-Index - 607

eISSN - 1524-4539

pISSN - 0009-7322

DOI - 10.1161/circulationaha.108.774752

Subject(s) - medicine , heart failure , cardiology , stimulation , endocrinology , myocyte , cardiac function curve , cardiac resynchronization therapy , adrenergic , receptor , ejection fraction

Cardiac resynchronization therapy (CRT) is the first clinical heart failure treatment that improves chamber systolic function in both the short-term and long-term yet also reduces mortality. The mechanical impact of CRT is immediate and well documented, yet its long-term influences on myocyte function and adrenergic modulation that may contribute to its sustained benefits are largely unknown.

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