Calcium-Activated Potassium Channels Contribute to Human Coronary Microvascular Dysfunction After Cardioplegic Arrest | Zendy

Jun Feng | Zendy; Yuhong Liu | Zendy; Richard Clements | Zendy; Neel R. Sodha | Zendy; Kamal R. Khabbaz | Zendy; Venkatachalam Senthilnathan | Zendy; Katherine K. Nishimura | Zendy; Seth L. Alper | Zendy; Frank W. Sellke | Zendy

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Calcium-Activated Potassium Channels Contribute to Human Coronary Microvascular Dysfunction After Cardioplegic Arrest

Author(s) -

Jun Feng,

Yuhong Liu,

Richard Clements,

Neel R. Sodha,

Kamal R. Khabbaz,

Venkatachalam Senthilnathan,

Katherine K. Nishimura,

Seth L. Alper,

Frank W. Sellke

Publication year - 2008

Publication title -

circulation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 7.795

H-Index - 607

eISSN - 1524-4539

pISSN - 0009-7322

DOI - 10.1161/circulationaha.107.755827

Subject(s) - medicine , cardiology , calcium , potassium , potassium channel , calcium activated potassium channel , chemistry , organic chemistry

Cardioplegic arrest (CP) followed by reperfusion after cardiopulmonary bypass induces coronary microvascular dysfunction. We investigated the role of calcium-activated potassium (K(Ca)) channels in this dysfunction in the human coronary microvasculature.

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