Laminin-α4 and Integrin-Linked Kinase Mutations Cause Human Cardiomyopathy Via Simultaneous Defects in Cardiomyocytes and Endothelial Cells | Zendy

Ralph Knöll | Zendy; Ruben Postel | Zendy; Jianming Wang | Zendy; Ralph Krätzner | Zendy; Gerrit Hennecke | Zendy; Andrei M. Vacaru | Zendy; Padmanabhan Vakeel | Zendy; Cornelia Schubert | Zendy; Kenton Murthy | Zendy; Brinda K. Rana | Zendy; Dieter Kube | Zendy; Gudrun Knöll | Zendy; Katrin Schäfer | Zendy; Takeharu Hayashi | Zendy; Torbjörn Holm | Zendy; Akinori Kimura | Zendy; Nicholas J. Schork | Zendy; Mohammad R. Toliat | Zendy; Peter Nürnberg | Zendy; HeinzPeter Schultheiss | Zendy; Wolfgang Schäper | Zendy; Jutta Schaper | Zendy; Erik Bos | Zendy; Jeroen den Hertog | Zendy; Fredericus J. M. van Eeden | Zendy; Peter J. Peters | Zendy; Gerd Hasenfuß | Zendy; Kenneth R. Chien | Zendy; Jeroen Bakkers | Zendy

Open Access

Laminin-α4 and Integrin-Linked Kinase Mutations Cause Human Cardiomyopathy Via Simultaneous Defects in Cardiomyocytes and Endothelial Cells

Author(s) -

Ralph Knöll,

Ruben Postel,

Jianming Wang,

Ralph Krätzner,

Gerrit Hennecke,

Andrei M. Vacaru,

Padmanabhan Vakeel,

Cornelia Schubert,

Kenton Murthy,

Brinda K. Rana,

Dieter Kube,

Gudrun Knöll,

Katrin Schäfer,

Takeharu Hayashi,

Torbjörn Holm,

Akinori Kimura,

Nicholas J. Schork,

Mohammad R. Toliat,

Peter Nürnberg,

HeinzPeter Schultheiss,

Wolfgang Schäper,

Jutta Schaper,

Erik Bos,

Jeroen den Hertog,

Fredericus J. M. van Eeden,

Peter J. Peters,

Gerd Hasenfuß,

Kenneth R. Chien,

Jeroen Bakkers

Publication year - 2007

Publication title -

circulation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 7.795

H-Index - 607

eISSN - 1524-4539

pISSN - 0009-7322

DOI - 10.1161/circulationaha.107.689984

Subject(s) - medicine , laminin , integrin , integrin linked kinase , cardiomyopathy , microbiology and biotechnology , kinase , protein kinase a , extracellular matrix , heart failure , cyclin dependent kinase 2 , biology , receptor

Extracellular matrix proteins, such as laminins, and endothelial cells are known to influence cardiomyocyte performance; however, the underlying molecular mechanisms remain poorly understood.

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