Hyperhomocysteinemia Alters Cardiac Substrate Metabolism by Impairing Nitric Oxide Bioavailability Through Oxidative Stress | Zendy

Nobuhiro Suematsu | Zendy; Caroline Ojaimi | Zendy; Shintaro Kinugawa | Zendy; Zipping Wang | Zendy; Xiaobin Xu | Zendy; Ákos Koller | Zendy; Fabio A. Recchia | Zendy; Thomas H. Hintze | Zendy

Open Access

Hyperhomocysteinemia Alters Cardiac Substrate Metabolism by Impairing Nitric Oxide Bioavailability Through Oxidative Stress

Author(s) -

Nobuhiro Suematsu,

Caroline Ojaimi,

Shintaro Kinugawa,

Zipping Wang,

Xiaobin Xu,

Ákos Koller,

Fabio A. Recchia,

Thomas H. Hintze

Publication year - 2007

Publication title -

circulation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 7.795

H-Index - 607

eISSN - 1524-4539

pISSN - 0009-7322

DOI - 10.1161/circulationaha.106.652693

Subject(s) - hyperhomocysteinemia , medicine , endocrinology , ascorbic acid , oxidative stress , nitric oxide , nitric oxide synthase , apocynin , cystathionine beta synthase , homocysteine , nadph oxidase , chemistry , biochemistry , methionine , food science , amino acid

Hyperhomocysteinemia (HHcy) has been considered a vascular disease associated with increased levels of oxidative stress that results in scavenging of NO. However, little is known of the impact of HHcy on cardiac function and especially myocardial metabolism.

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