Increased Expression of Syndecan-1 Protects Against Cardiac Dilatation and Dysfunction After Myocardial Infarction | Zendy

Davy Vanhoutte | Zendy; Mark W.M. Schellings | Zendy; Martin Götte | Zendy; Melissa Swinnen | Zendy; Veronica Hérias | Zendy; Martin K. Wild | Zendy; Dietmar Vestweber | Zendy; Emmanuel Chorianopoulos | Zendy; Vı́ctor Cortés | Zendy; Attilio Rigotti | Zendy; Mary Ann Stepp | Zendy; Frans Van de Werf | Zendy; Peter Carmeliet | Zendy; Yigal M. Pinto | Zendy; Stéphane Heymans | Zendy

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Increased Expression of Syndecan-1 Protects Against Cardiac Dilatation and Dysfunction After Myocardial Infarction

Author(s) -

Davy Vanhoutte,

Mark W.M. Schellings,

Martin Götte,

Melissa Swinnen,

Veronica Hérias,

Martin K. Wild,

Dietmar Vestweber,

Emmanuel Chorianopoulos,

Vı́ctor Cortés,

Attilio Rigotti,

Mary Ann Stepp,

Frans Van de Werf,

Peter Carmeliet,

Yigal M. Pinto,

Stéphane Heymans

Publication year - 2007

Publication title -

circulation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 7.795

H-Index - 607

eISSN - 1524-4539

pISSN - 0009-7322

DOI - 10.1161/circulationaha.106.644609

Subject(s) - medicine , inflammation , myocardial infarction , wound healing , syndecan 1 , cardiac function curve , heart failure , cardiology , endocrinology , immunology , cell , biology , genetics

The cell-associated proteoglycan syndecan-1 (Synd1) closely regulates inflammation and cell-matrix interactions during wound healing and tumorigenesis. The present study investigated whether Synd1 may also regulate cardiac inflammation, matrix remodeling, and function after myocardial infarction (MI).

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