Transcriptional Profiling in Coronary Artery Disease | Zendy

Thomas W. Chittenden | Zendy; Jonathan A. Sherman | Zendy; Fei Xiong | Zendy; Amy Hall | Zendy; Anthony A. Lanahan | Zendy; Jennifer M. Taylor | Zendy; Hangjun Duan | Zendy; Justin D. Pearlman | Zendy; Jason H. Moore | Zendy; Stephen M. Schwartz | Zendy; Michael Simons | Zendy

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Transcriptional Profiling in Coronary Artery Disease

Author(s) -

Thomas W. Chittenden,

Jonathan A. Sherman,

Fei Xiong,

Amy Hall,

Anthony A. Lanahan,

Jennifer M. Taylor,

Hangjun Duan,

Justin D. Pearlman,

Jason H. Moore,

Stephen M. Schwartz,

Michael Simons

Publication year - 2006

Publication title -

circulation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 7.795

H-Index - 607

eISSN - 1524-4539

pISSN - 0009-7322

DOI - 10.1161/circulationaha.106.628396

Subject(s) - medicine , collateral circulation , coronary artery disease , arteriogenesis , gene expression profiling , monocyte , collateral , angiogenesis , transcriptome , cardiology , gene expression , genetics , biology , gene , finance , economics

The development of collateral circulation plays an important role in protecting tissues from ischemic damage, and its stimulation has emerged as one of principal approaches to therapeutic angiogenesis. Clinical observations have documented substantial differences in the extent of collateralization among patients with coronary artery disease (CAD), with some individuals demonstrating marked abundance and others showing nearly complete absence of these vessels. Recent studies have suggested that circulating monocytes play a major role in collateral growth. The present study was undertaken to determine transcriptional profiles of circulating monocytes in CAD patients with different extents of collateral growth.

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