Cardiac S100A1 Protein Levels Determine Contractile Performance and Propensity Toward Heart Failure After Myocardial Infarction | Zendy

Patrick Most | Zendy; Hanna Seifert | Zendy; Erhe Gao | Zendy; Hajime Funakoshi | Zendy; Mirko Völkers | Zendy; Jörg Heierhorst | Zendy; Andrew Remppis | Zendy; Sven T. Pleger | Zendy; Brent R. DeGeorge | Zendy; Andrea D. Eckhart | Zendy; Arthur M. Feldman | Zendy; Walter J. Koch | Zendy

Open Access

Cardiac S100A1 Protein Levels Determine Contractile Performance and Propensity Toward Heart Failure After Myocardial Infarction

Author(s) -

Patrick Most,

Hanna Seifert,

Erhe Gao,

Hajime Funakoshi,

Mirko Völkers,

Jörg Heierhorst,

Andrew Remppis,

Sven T. Pleger,

Brent R. DeGeorge,

Andrea D. Eckhart,

Arthur M. Feldman,

Walter J. Koch

Publication year - 2006

Publication title -

circulation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 7.795

H-Index - 607

eISSN - 1524-4539

pISSN - 0009-7322

DOI - 10.1161/circulationaha.106.622415

Subject(s) - medicine , heart failure , cardiology , myocardial infarction , dilated cardiomyopathy , cardiac function curve , endocrinology

Diminished cardiac S100A1 protein levels are characteristic of ischemic and dilated human cardiomyopathy. Because S100A1 has recently been identified as a Ca2+-dependent inotropic factor in the heart, this study sought to explore the pathophysiological relevance of S100A1 levels in development and progression of postischemic heart failure (HF).

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