Acute Neurocardiogenic Injury After Subarachnoid Hemorrhage | Zendy

Nader M. Banki | Zendy; Alexander Kopelnik | Zendy; Michael W. Dae | Zendy; Jacob C. Miss | Zendy; Poyee P. Tung | Zendy; Michael T. Lawton | Zendy; Barbara J. Drew | Zendy; Elyse Foster | Zendy; Wade S. Smith | Zendy; William W. Parmley | Zendy; Jonathan G. Zaroff | Zendy

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Acute Neurocardiogenic Injury After Subarachnoid Hemorrhage

Author(s) -

Nader M. Banki,

Alexander Kopelnik,

Michael W. Dae,

Jacob C. Miss,

Poyee P. Tung,

Michael T. Lawton,

Barbara J. Drew,

Elyse Foster,

Wade S. Smith,

William W. Parmley,

Jonathan G. Zaroff

Publication year - 2005

Publication title -

circulation

Language(s) - English

Resource type - Journals

eISSN - 1524-4539

pISSN - 0009-7322

DOI - 10.1161/circulationaha.105.558239

Subject(s) - medicine , cardiology , pathophysiology , perfusion , subarachnoid hemorrhage , scintigraphy , ejection fraction , ischemia , sympathetic denervation , sympathectomy , troponin , denervation , myocardial infarction , heart failure

Left ventricular (LV) systolic dysfunction has been reported in humans with subarachnoid hemorrhage (SAH), and its underlying pathophysiology remains controversial. Possible mechanisms include myocardial ischemia versus excessive catecholamine release from sympathetic nerve terminals.

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